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This deck focuses on 10a Environmental Justice Health Risk, giving you a quick way to review the definitions, rules, and examples that matter most for MCAT Psychological Social Foundations.
Study 10a Environmental Justice Health Risk in MCAT Psychological Social Foundations with focused flashcards that help you recognize the idea, recall the key rule, and apply it in practice-style prompts.
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What is a hazard in environmental health?
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An agent or condition that can cause harm. Identifies potential sources of injury or illness in environmental contexts, essential for risk assessment.
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This deck focuses on 10a Environmental Justice Health Risk, giving you a quick way to review the definitions, rules, and examples that matter most for MCAT Psychological Social Foundations.
Work through these flashcards in short sessions. Try to answer each prompt before flipping the card, then revisit any cards you miss until the explanation feels automatic.
Answer: An agent or condition that can cause harm. Identifies potential sources of injury or illness in environmental contexts, essential for risk assessment.
Answer: Differential susceptibility. Accounts for how limited resources and pre-existing conditions amplify health impacts from identical hazard levels.
Answer: Contact between a person and a hazard (dose over time). Measures the interaction intensity and duration, crucial for determining potential health impacts in populations.
Answer: Not In My Back Yard; hazards displaced toward less powerful communities. Wealthy areas reject hazards, concentrating them in poor communities.
Answer: Exposure is contact; dose is amount absorbed by the body. Exposure is external contact; dose measures internal absorption.
Answer: Epidemiologic observational study (often cohort or cross-sectional). Observes natural exposure patterns without experimental manipulation.
Answer: Change in effect magnitude as dose increases. Demonstrates the correlation between increasing toxin levels and escalating health effects in exposed individuals.
Answer: Area with limited access to affordable, nutritious food. Lack of healthy food options increases diet-related disease risk.
Answer: Structural racism (in environmental siting and regulation). Encompasses institutional practices that disproportionately site hazards in communities of color, driven by unequal power dynamics.
Answer: Social determinants of health. Encompass upstream factors like access to green spaces and pollution levels that influence community health trajectories.
Answer: Any nonzero dose carries some risk. Assumes linear risk increase with dose, used for substances where even minimal exposure may initiate cancer.
Answer: Cumulative risk (cumulative exposure). Integrates multiple environmental and social stressors to assess overall health risk in communities.
Answer: Synergistic interaction of exposures. Describes amplified health detriments when psychosocial stressors exacerbate toxicological effects.
Answer: Act to prevent harm despite incomplete scientific certainty. Prioritizes prevention when full evidence of harm is still developing.
Answer: Contact with an agent over time (e.g., pollutant dose or concentration). Quantifies contact intensity and duration for risk assessment.
Answer: Fair treatment and meaningful involvement in environmental health policies. Ensures all communities have equal voice in environmental decisions affecting their health.
Answer: Unequal contact with hazards across groups, often by race or SES. Low-income and minority groups face more environmental hazards than others.
Answer: Spatial separation of groups that concentrates hazards and limits resources. Historical policies created unequal exposure to environmental hazards.
Answer: Inequitable siting (environmental injustice in facility placement). Powerful communities resist hazards, pushing them to vulnerable areas.
Answer: Cumulative burden (allostatic load conceptually). Reflects the long-term accumulation of stressors, similar to physiological wear from chronic adversity.
Answer: An unfair, avoidable health difference produced by systemic disadvantage. Emphasizes injustice and systemic causes, not just differences.
Answer: Ingestion of lead-contaminated dust or paint chips. Occurs primarily through hand-to-mouth behavior in deteriorating structures, common in low-income housing.
Answer: Disproportionate environmental burdens on racial or ethnic minority groups. Minority communities bear unfair share of pollution and environmental hazards.
Answer: Higher rates of asthma and other cardiopulmonary disease. Air pollution triggers respiratory inflammation and cardiovascular stress.
Answer: Differential susceptibility due to comorbidities, stress, and limited healthcare. Social stressors amplify biological vulnerability to pollutants.
Answer: Environmental injustice. Highlights systemic inequities where vulnerable populations bear disproportionate environmental harms without fair benefits.
Answer: Systematic differences in health outcomes across population groups. Reflects preventable, unjust differences in health between groups.
Answer: Exposure may contribute to disease; confounding must still be evaluated. Association exists but causation requires ruling out other factors.
Answer: Higher dose is associated with greater biological effect or disease risk. Shows proportional increase in harm with increasing exposure levels.
Answer: Cumulative risk (cumulative impact). Multiple stressors interact synergistically to worsen health outcomes.
Answer: Fair distribution of environmental benefits and burdens across groups. Ensures equitable access to clean environments and protection from hazards, addressing disparities in public health outcomes.
Answer: Environmental inequality. Describes systematic differences in hazard exposure based on social factors.
Answer: Spatial separation by race/SES that concentrates hazards in some neighborhoods. Segregation clusters environmental hazards in minority neighborhoods.
Answer: Human-made surroundings (housing, roads, industry) shaping exposures and health. Physical infrastructure influences exposure pathways and health outcomes.
Answer: Differential exposure. Explains how socioeconomic status and geographic location result in higher contact with environmental toxins.
Answer: Disproportionate environmental burdens on racial or ethnic minority groups. Intentional or systemic placement of hazards in minority communities.
Answer: Smoking prevalence (or secondhand smoke exposure) in the population. Tobacco smoke independently causes asthma, potentially masking traffic effects.
Answer: Disproportionate air and noise pollution exposure. Results from infrastructure decisions that exacerbate respiratory and cardiovascular risks in underserved neighborhoods.
Answer: No adverse effect is expected below a certain dose. Posits a safe exposure level exists, guiding regulatory standards for non-carcinogenic substances.
Answer: Food swamp. Promotes poor dietary choices by saturating areas with fast food, exacerbating health disparities in vulnerable communities.
Answer: Urban warming from surfaces; higher heat risk in low-tree, low-income areas. Concrete absorbs heat; poor areas lack cooling green spaces.
Answer: Cumulative risk (or cumulative burden). Multiple stressors compound health risks beyond individual effects.
Answer: A preventable health difference tied to social, economic, or environmental disadvantage. Links health differences to modifiable social factors, not just biology.
Answer: Stricter emission controls and equitable enforcement in high-burden areas. Targets pollution sources and ensures fair regulatory protection.
Answer: An area with limited access to affordable, nutritious food. Geographic barrier to healthy eating, often in low-income areas.
Answer: Opposition to local hazards that shifts burdens onto other communities. "Not In My Back Yard" - pushes hazards to less powerful communities.
Answer: Increasing concentration of a toxin at higher trophic levels. Toxins accumulate up food chains, concentrating in top predators.
Answer: Amount of a hazard absorbed by the body. Represents the internalized quantity of a toxin, influencing the severity of biological responses.
Answer: Probability of adverse health effects from an environmental hazard. Quantifies the likelihood of harm based on exposure to pollutants or conditions in the environment.
Answer: Fair treatment and meaningful involvement in environmental decisions. Ensures all communities have a voice in environmental policies affecting them.
Answer: Children (developing nervous system). Rapid brain development makes them highly sensitive to neurotoxins, leading to cognitive and behavioral impairments.
Answer: Residential proximity to pollution sources due to housing constraints. Links economic disadvantage to increased hazard proximity, perpetuating health inequities in low-income areas.
Answer: A systematic difference in health outcomes between population groups. Arises from social, economic, and environmental factors leading to unequal health burdens among demographic groups.
Answer: A person's subjective judgment of the likelihood and severity of harm. Influenced by personal experience, culture, and trust in institutions.
Answer: Social determinants of health. Recognizes that health outcomes depend on living conditions and environment.
Answer: Equity allocates resources by need; equality allocates the same to all. Equity addresses existing disparities; equality ignores different starting points.
Answer: Food desert. Contributes to nutritional deficiencies and obesity by limiting access to fresh produce in underserved areas.
Answer: Institutional policies creating unequal access to resources and protections. Systems and policies perpetuate unequal environmental protections.
Answer: Exposure is contact; dose is absorbed amount. Distinguishes external contact from internal uptake, critical for accurate toxicological evaluations.
Answer: Human-made surroundings that shape exposure and behavior (housing, roads, parks). Physical infrastructure influences exposure patterns and health behaviors.
Answer: Particulate matter (especially PM2.5) and ozone. These fine particles and ground-level gas penetrate lungs, triggering inflammation and worsening respiratory conditions.
Answer: Historic denial of loans by area, promoting disinvestment and later hazard siting. Discriminatory lending created vulnerable areas for hazardous facilities.