What this deck covers
This deck focuses on 7a Brain Neurotransmitters Behavior, giving you a quick way to review the definitions, rules, and examples that matter most for MCAT Psychological Social Foundations.
Study 7a Brain Neurotransmitters Behavior in MCAT Psychological Social Foundations with focused flashcards that help you recognize the idea, recall the key rule, and apply it in practice-style prompts.
0% Complete
Which neurotransmitter is the primary excitatory transmitter in the central nervous system?
Tap card or press Space to flip
Glutamate. Opens cation channels to depolarize postsynaptic neurons.
How well did you know it?
Card 1 / 291
Space to flip · ← / → to move · once flipped, → Got it · ← Still learning
This deck focuses on 7a Brain Neurotransmitters Behavior, giving you a quick way to review the definitions, rules, and examples that matter most for MCAT Psychological Social Foundations.
Work through these flashcards in short sessions. Try to answer each prompt before flipping the card, then revisit any cards you miss until the explanation feels automatic.
Answer: Glutamate. Opens cation channels to depolarize postsynaptic neurons.
Answer: Somatosensory processing and spatial attention; integrates sensory input. The parietal lobe handles tactile sensations and orients attention in space by merging multisensory data.
Answer: Autonomic vital functions (breathing, heart rate). Located in the lower brainstem, it controls essential life-sustaining reflexes.
Answer: Executive control: planning, inhibition, working memory, decision-making. Located in frontal lobe, controls higher-order cognitive functions.
Answer: Language comprehension (receptive language). Located in left temporal lobe; damage causes receptive aphasia.
Answer: Norepinephrine. Released during stress and fight-or-flight responses.
Answer: Thalamus. Acts as the brain's sensory switchboard, routing signals to appropriate cortical areas.
Answer: Hypothalamus. Links nervous and endocrine systems, maintaining body's internal balance.
Answer: Broca area. Damage causes expressive aphasia with impaired speech fluency.
Answer: Hippocampus. Part of temporal lobe; converts short-term to long-term memories.
Answer: Language comprehension (receptive language). Located in left temporal lobe; damage causes receptive aphasia.
Answer: Speech production (expressive language). Broca's area in the frontal lobe coordinates the articulatory muscles necessary for verbal expression.
Answer: Sensory relay to cortex (except olfaction); also arousal/attention. The thalamus gates sensory information to the cortex while regulating levels of consciousness.
Answer: Language comprehension (receptive language). Wernicke's area in the temporal lobe decodes auditory input to facilitate understanding of spoken language.
Answer: Medulla oblongata. Brainstem region containing vital centers for survival functions.
Answer: Glutamate. Opens ion channels causing depolarization and neuronal firing.
Answer: Midbrain. Contains substantia nigra and regulates consciousness levels.
Answer: Initiation/modulation of movement and habit (procedural) learning. Subcortical nuclei that select and inhibit motor programs.
Answer: Dopamine. Depletion in substantia nigra causes motor symptoms of Parkinson's.
Answer: Connects cerebral hemispheres for interhemispheric communication. Largest white matter tract enabling hemispheric coordination.
Answer: Occipital lobe. Processes visual information from the eyes at the back of the brain.
Answer: Homeostasis; autonomic control; drives; pituitary regulation. Links nervous and endocrine systems; maintains body balance.
Answer: Autonomic control of breathing, heart rate, and blood pressure. Part of brainstem, controls vital life functions.
Answer: Motor coordination and procedural learning. Fine-tunes movements and stores implicit motor memories.
Answer: Homeostasis; drives; controls pituitary/endocrine output. Links nervous and endocrine systems via pituitary control.
Answer: Processes touch, pain, temperature, and proprioception. Located in postcentral gyrus; receives sensory input.
Answer: Homeostasis and endocrine control via the pituitary (drives, ANS). Links nervous and endocrine systems to maintain body equilibrium.
Answer: Glutamate. Activates AMPA and NMDA receptors to depolarize postsynaptic neurons.
Answer: Threat detection and fear/aggression-related emotional learning. Part of limbic system that processes emotional significance of stimuli.
Answer: Coordination of movement and motor learning. Fine-tunes motor commands for smooth, precise movements.
Answer: Serotonin (5-HT). SSRIs block reuptake to increase synaptic serotonin levels.
Answer: Fear/threat processing and emotional learning. Rapidly detects threats and triggers emotional responses.
Answer: Glutamate. Depolarizes neurons, increasing action potential likelihood.
Answer: Regulates arousal/alertness and sleep-wake transitions. Brainstem network that maintains consciousness and attention.
Answer: GABA. Hyperpolarizes neurons; enhanced by benzodiazepines and alcohol.
Answer: Initiates voluntary skeletal muscle movement. Located in precentral gyrus, sends signals to spinal motor neurons.
Answer: Coordination, balance, motor learning, movement timing. Fine-tunes motor commands for smooth, precise movements.
Answer: Hippocampus (medial temporal lobe). Damage prevents formation of new declarative memories (H.M. case).
Answer: Major excitatory neurotransmitter; increases neuronal firing. Opens sodium channels, depolarizing neurons to trigger action potentials.
Answer: Language comprehension (receptive language). Left temporal lobe area; damage causes receptive aphasia.
Answer: Emotion processing, especially fear and threat detection. Part of limbic system that rapidly evaluates stimuli for danger.
Answer: Amygdala. Part of limbic system; triggers fight-or-flight responses.
Answer: Mood, sleep, and appetite regulation; implicated in depression/anxiety. Synthesized from tryptophan; low levels linked to mood disorders.
Answer: Dopamine deficiency (nigrostriatal pathway). Loss of dopaminergic neurons in substantia nigra causes motor symptoms.
Answer: Arousal, vigilance, attention; sympathetic stress response. Fight-or-flight neurotransmitter; enhances focus under stress.
Answer: Hippocampus. Part of limbic system, converts short-term to long-term memories.
Answer: Hippocampus. Converts short-term memories to long-term storage, especially facts and events.
Answer: Glutamate. Opens Na+ channels, depolarizing neurons.
Answer: Arousal, alertness, and sleep-wake regulation. Network of neurons maintaining consciousness and sleep cycles.
Answer: Connects left and right cerebral hemispheres. Largest white matter tract enabling interhemispheric communication.
Answer: Executive functions: planning, inhibition, working memory, decision-making. Located in frontal lobe, controls higher-order cognitive processes.
Answer: Processes touch, pain, temperature, and proprioception. Located in parietal lobe along postcentral gyrus.
Answer: Vital functions: breathing, heart rate, arousal, reflexes. Controls autonomic functions essential for survival.
Answer: Fear processing and emotional salience. Detects threats and assigns emotional significance to stimuli.
Answer: Connects left and right cerebral hemispheres. Largest white matter tract enabling interhemispheric communication.
Answer: HPA axis. Hypothalamic-pituitary-adrenal axis releases cortisol under stress.
Answer: Frontal lobe. Contains motor strip (precentral gyrus) that initiates voluntary movements.
Answer: Glutamate. Opens ion channels causing depolarization and neural firing.
Answer: Frontal lobe (prefrontal cortex). Controls planning, decision-making, and behavioral inhibition.
Answer: Adrenal cortex. HPA axis activation triggers cortisol release during stress response.
Answer: Executive functions: planning, inhibition, decision-making. Located in prefrontal cortex, controls higher-order cognitive processes.
Answer: Sympathetic: fight-or-flight; parasympathetic: rest-and-digest. These autonomic divisions opposingly regulate physiological responses to stress and recovery.
Answer: Acetylcholine. Binds nicotinic receptors to trigger muscle fiber depolarization.
Answer: Acetylcholine. Also important in memory and attention; decreased in Alzheimer's.
Answer: Thalamus. Acts as sensory gateway; olfaction bypasses it directly to cortex.
Answer: GABA. Opens chloride channels causing hyperpolarization and reduced firing.
Answer: Medulla oblongata. Contains vital centers for autonomic functions.
Answer: Opioid peptides; analgesia and euphoria. Body's natural painkillers that bind opioid receptors.
Answer: Acetylcholine. Binds nicotinic receptors to depolarize muscle fibers.
Answer: Arousal, wakefulness, and sleep-wake transitions. The reticular formation modulates brainstem activity to control consciousness and alertness.
Answer: Hippocampus (medial temporal lobe). Bilateral damage prevents formation of new declarative memories.
Answer: Wernicke area lesion. Produces fluent but meaningless speech (receptive aphasia).
Answer: Substantia nigra. Produces dopamine; degeneration causes tremor and movement difficulties.
Answer: Reticular activating system (brainstem reticular formation). Network of neurons that maintains consciousness and alertness.
Answer: Initiates voluntary skeletal muscle movement. Located in precentral gyrus; controls contralateral movement.
Answer: Dopamine. Released in reward pathways; dysfunction linked to Parkinson's and schizophrenia.
Answer: Norepinephrine. Released by adrenal medulla and locus coeruleus.
Answer: Major inhibitory neurotransmitter in the CNS. Reduces neural excitability; dysfunction causes seizures/anxiety.
Answer: Processes touch, pain, temperature, and proprioception. Located in parietal lobe, receives sensory input from body.
Answer: Sensory relay to cortex (except olfaction). Acts as a gateway filtering sensory information before reaching cortex.
Answer: Occipital lobe. Processes visual information at the back of the brain.
Answer: Cerebellum. Located at the back of the brain, it smooths and coordinates movements.
Answer: Movement initiation and habit/procedural learning. Includes caudate, putamen, and globus pallidus for motor control.
Answer: Hippocampus. Part of the temporal lobe critical for converting short-term to long-term memory.
Answer: Language comprehension (receptive language). Located in temporal lobe, damage causes receptive aphasia.
Answer: Reward/motivation and motor control; implicated in addiction and Parkinson disease. Key neurotransmitter in reward pathways and voluntary movement.
Answer: Sleep/arousal and relay to cerebellum. Contains nuclei for REM sleep and connects to cerebellum.
Answer: Acetylcholine. Binds nicotinic receptors on muscle fibers to initiate contraction.
Answer: Connects the left and right hemispheres. Largest white matter tract enabling interhemispheric communication.
Answer: Connects left and right cerebral hemispheres for interhemispheric transfer. Largest white matter tract enabling hemispheric communication.
Answer: Serotonin. Low levels associated with major depressive disorder; SSRIs increase its availability.
Answer: Consolidation of declarative memories; spatial navigation. Forms new explicit memories and cognitive maps of environment.
Answer: Temporal lobe. Houses Wernicke's area for understanding spoken language.
Answer: Major inhibitory neurotransmitter; increases inhibition (hyperpolarizes). GABA binds to receptors that promote chloride influx, reducing neuronal firing rates.
Answer: Initiation and modulation of movement; habit learning and reward. The basal ganglia modulate motor patterns and reinforce behaviors through reward-based learning.
Answer: Occipital lobe. Processes all visual information from the eyes.
Answer: Primary inhibitory neurotransmitter in the CNS. Reduces neural activity; benzodiazepines enhance its effect.
Answer: GABA. Hyperpolarizes neurons to reduce neural excitability.
Answer: Relay station for sensory signals to cortex (except olfaction). All sensory pathways synapse here before reaching cortex.
Answer: Major inhibitory neurotransmitter; decreases neuronal firing. Opens chloride channels, hyperpolarizing neurons to prevent firing.