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This deck focuses on Respiratory Distress And Failure, giving you a quick way to review the definitions, rules, and examples that matter most for NREMT AEMT Level.
Study Respiratory Distress And Failure in NREMT AEMT Level with focused flashcards that help you recognize the idea, recall the key rule, and apply it in practice-style prompts.
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Which device is preferred for a spontaneously breathing patient needing high oxygen concentration?
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Nonrebreather mask. Provides high FiO2 up to 100% by using a reservoir bag and one-way valves to minimize room air entrainment.
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This deck focuses on Respiratory Distress And Failure, giving you a quick way to review the definitions, rules, and examples that matter most for NREMT AEMT Level.
Work through these flashcards in short sessions. Try to answer each prompt before flipping the card, then revisit any cards you miss until the explanation feels automatic.
Answer: Nonrebreather mask. Provides high FiO2 up to 100% by using a reservoir bag and one-way valves to minimize room air entrainment.
Answer: Accessory muscle use or retractions. These physical signs reflect the body's effort to recruit additional muscles to overcome increased respiratory resistance or decreased lung compliance.
Answer: Increased work of breathing with adequate ventilation and oxygenation. This definition captures the compensatory phase where patients exhibit labored breathing but maintain sufficient gas exchange without immediate decompensation.
Answer: Systemic corticosteroid (for example, methylprednisolone). Inhibits inflammatory mediators to reduce airway edema and mucus production in exacerbations.
Answer: Crackles (rales). Discontinuous sounds produced by air bubbling through alveolar fluid, indicating pulmonary edema or pneumonia.
Answer: Inhaled beta-2 agonist (for example, albuterol). Stimulates beta-2 receptors to relax bronchial smooth muscle, rapidly relieving acute airway obstruction.
Answer: Suspected basilar skull fracture or severe midface trauma. Risks inadvertent intracranial placement through cribriform plate defects, potentially causing brain injury or infection.
Answer: Inadequate breathing (poor rate, depth, or tidal volume). Assisted ventilation is required when spontaneous breathing fails to achieve adequate minute volume for gas exchange.
Answer: Wheezing. High-pitched expiratory sounds result from turbulent airflow through constricted lower airways due to smooth muscle contraction.
Answer: Elevated arterial carbon dioxide level. Indicates ventilatory failure where CO2 retention occurs, typically above 45 mmHg, leading to respiratory acidosis.
Answer: CPAP, if not contraindicated. Positive end-expiratory pressure recruits alveoli, reduces work of breathing, and decreases cardiac preload.
Answer: Nasopharyngeal airway (NPA), if no contraindication. Tolerated in patients with intact gag reflex as it does not stimulate the oropharynx, maintaining patency in altered mental status.
Answer: Wheezing with hypotension and urticaria or angioedema. Systemic allergic response triggers mast cell degranulation, causing bronchoconstriction and vasodilatory shock.
Answer: Diminished or absent breath sounds. Reflects profound airflow limitation, such as in pneumothorax or severe asthma, where minimal ventilation reaches lung fields.
Answer: Naloxone with ventilatory support as needed. Opioid antagonist reverses central respiratory depression by competing for mu-receptors, restoring ventilatory drive.
Answer: Venturi mask. Allows delivery of controlled FiO2 percentages to prevent oxygen-induced hypercapnia in patients with chronic hypoxic drive.
Answer: Decreasing mental status (fatigue with poor respiratory effort). This indicates respiratory muscle exhaustion, signaling progression from distress to failure with imminent risk of arrest.
Answer: Low arterial oxygen level. Refers to insufficient oxygen partial pressure in arterial blood, often below 60 mmHg, compromising tissue oxygenation.
Answer: Severe dyspnea with absent unilateral breath sounds and hypotension. Combination indicates intrathoracic pressure buildup compressing lung and shifting mediastinum, impairing venous return.
Answer: Inadequate ventilation or oxygenation, or both, with impending arrest risk. This describes the decompensated state where gas exchange fails, leading to potential cardiopulmonary arrest if not addressed promptly.
Answer: Troubleshoot pulse oximeter accuracy before escalating therapy. Poor plethysmograph suggests artifact or poor perfusion, making SpO2 unreliable despite clinical stability.
Answer: Asthma is often reversible bronchospasm; COPD is chronic airflow limitation. Asthma involves episodic, reversible obstruction from inflammation; COPD features persistent structural damage limiting airflow.
Answer: Stridor. Inspiratory crowing noise from turbulent flow through narrowed upper airway, often due to edema or obstruction.
Answer: Pulmonary edema (often cardiogenic). Increased hydrostatic pressure forces fluid into alveoli, producing characteristic sputum and auscultatory findings.